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Glutathione and mitochondria determine acute defense responses and adaptive processes in cadmium-induced oxidative stress and toxicity of the kidney

GGsTop treatment did not affect levels of GSH, GSSG or glutamate in tumours (Extended Data Fig

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Additionally, the transient accumulation of H 2 O 2 following pathogen infection leads to localized programmed cell death or hypersensitive (HR) response and stimulates crosslinking of cell wall proteins, thereby preventing pathogen spread in the plant (Grant and Loake, Plants have an efficient anti-oxidant defense system which scavenges the excess ROS produced in the cell under different oxidative stress conditions